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What is L.V.G and why is Detoxing Important

Writer: Healthy FoxHealthy Fox

Updated: Feb 8

“I found out my bile detox pathway was dysfunctional because of one silly thing, I burped a lot… seriously. After fat heavy meals I never knew why I always burped, Dr. Michelle explained that basically your liver and gallbladder produce bile in order to break down fats. If it’s not working properly, one of the first signs is burping (amongst other things)! Long term it can cause digestive issues, serious nutritional deficiencies and even be a serious medical problem. To add to it, this bile system is an important part of the detoxification process in order to process certain unwanted compounds out of the body. Pretty shortly after my burping is gone and after a few months never returned!” Bryce





L.V.G Complex™ – Comprehensive Support for Liver and Gallbladder Health


L.V.G Complex™ is a powerful blend of nutrients and herbal extracts designed to support liver and gallbladder function. By providing lipotropic substances, it aids in the elimination of fatty substances from the liver and promotes proper bile flow.* The ingredients in L.V.G Complex™ also support detoxification pathways and enhance the digestion and absorption of dietary fats and fat-soluble nutrients. This supplement is ideal for individuals needing liver support, those who have had their gallbladder removed, and those with skin issues that may benefit from improved fat absorption, particularly essential fatty acids.


Key Benefits:

  • Supports healthy liver and gallbladder function

  • Promotes proper bile flow and fat digestion

  • Assists in detoxification and nutrient absorption

  • Beneficial for those with skin issues related to fat digestion


Key Ingredients:

  • Methionine, Vitamin B12, and Vitamin B6: These nutrients play essential roles in methylation, which is crucial for effective detoxification. Methionine and vitamin B6 also help maintain healthy glutathione levels, which support liver health.*1

  • Milk Thistle (Silybum marianum): Known for its hepatoprotective properties, milk thistle has been shown to promote liver cell regeneration and protect against liver damage from various toxins, including alcohol and acetaminophen. It also helps enhance bile flow and may reduce inflammation, potentially increasing insulin sensitivity.*2,3,4

  • Inositol: In studies on non-alcoholic fatty liver disease (NAFLD), inositol has been shown to reduce liver fat accumulation and improve liver cell integrity by increasing glutathione levels and reducing oxidative stress.*6,7

  • Artichoke (Cynara scolymus): Artichoke extract has been shown to enhance glutathione activity and decrease reactive oxygen species. It also helps reduce liver enzyme levels and supports bile production, which aids in digestion and liver regeneration.*8,9

  • Beetroot (Beta vulgaris): A rich source of betaine, beetroot helps reduce liver fat accumulation and supports healthy bile flow. Studies have demonstrated its ability to decrease oxidative stress, inflammation, and lipid peroxidation in liver cells, providing additional hepatoprotective benefits.*10,11,12

  • Taurine: Taurine plays a key role in bile synthesis and is important for healthy liver and gallbladder function. It supports the conversion of cholesterol into bile acids, which are essential for fat digestion and absorption.*17,18

  • Ox Bile: Traditionally used in liver and biliary support, ox bile has been shown to help with fat digestion and absorption, particularly in individuals with impaired bile flow, such as those without a gallbladder or those with conditions like short bowel syndrome.*19-22

Gallbladder Support:

Milk thistle and artichoke leaf extract act as cholagogues, promoting bile flow and assisting in the elimination of toxins. Taurine also supports bile synthesis, and ox bile enhances fat digestion and absorption, benefiting those with low bile production. These ingredients help improve the digestive process, particularly for individuals with compromised gallbladder function.*17,18,19-22

Who Should Take L.V.G Complex™?

L.V.G Complex™ is beneficial for:

  • Individuals without a gallbladder

  • Those needing to support liver and gallbladder function

  • People who experience difficulty digesting fatty foods

  • Those with bloating, gas, gastrointestinal distress, or skin issues related to fat digestion

  • Individuals seeking detoxification support

Who Should Not Take L.V.G Complex™?

Patients experiencing acute upper abdominal pain or known bile duct obstruction should not use L.V.G Complex™.

References

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  2. Bijak M. Molecules. 2017;22(11):1942. doi:10.3390/molecules22111942.

  3. Federico A, et al. Molecules. 2017;22(2):191. doi:10.3390/molecules22020191.

  4. Vargas‑Mendoza N. World J Hepatol. 2014;6(3):144. doi:10.4254/wjh.v6.i3.144.

  5. Abenavoli L, et al. Phytother Res. 2010;24(10):1423‑1432. doi:10.1002/ptr.3207.

  6. Gillessen A, Schmidt HHJ. Adv Ther. 2020;37(4):1279‑1301. doi:10.1007/s12325‑020‑01251‑y.

  7. Faulstich H, et al. Arzneimittelforschung. 1980;30(3):452‑454.

  8. Hodges RE, Minich DM. J Nutr Metab. 2015;2015:1‑23. doi:10.1155/2015/760689.

  9. Atkuri KR, et al. Curr Opin Pharmacol. 2007;7(4):355‑359. doi:10.1016/j.coph.2007.04.005.

  10. Dehmlow C, et al. Hepatology. 1996;23(4):749‑754. doi:10.1002/hep.510230415.

  11. Lieber CS. Am J Clin Nutr. 2002;76(5):1183S‑1187S. doi:10.1093/ajcn/76/5.1183S.

  12. Feld JJ, et al. Gastroenterology. 2011;140(3):830‑839. doi:10.1053/j.gastro.2010.09.010.

  13. Saénz Rodriguez T, et al. Phytomedicine. 2002;9(8):687‑693. doi:10.1078/094471102321621278.

  14. Rangboo V, et al. Int J Hepatol. 2016;2016:1‑6. doi:10.1155/2016/4030476.

  15. Srivastava S, et al. Curr Res Nutr Food Sci J. 2019;7(1):96‑101. doi:10.12944/CRNFSJ.7.1.10.

  16. Krajka‑Kuźniak V, et al. Food Chem Toxicol. 2012;50(6):2027‑2033. doi:10.1016/j.fct.2012.03.062.

  17. Krajka‑Kuźniak V, et al. Br J Nutr. 2013;110(12):2138‑2149. doi:10.1017/S0007114513001645.

  18. Albasher G, et al. Biomolecules. 2019;9(7):261. doi:10.3390/biom9070261.

  19. Wang DQH. World J Gastroenterol. 2014;20(29):9952. doi:10.3748/wjg.v20.i29.9952.

  20. Gruy‑Kapral C, et al. Gastroenterology. 1999;116(1):15‑21. doi:10.1016/S0016‑5085(99)70223‑4.

  21. Little KH, et al. Dig Dis Sci. 1992;37(6):929‑933. doi:10.1007/BF01300393.

  22. Duszka K. Cells. 2022;11(15):2337. doi:10.3390/cells11152337.

  23. Guo J, et al. Lipids Health Dis. 2017;16(1):56. doi:10.1186/s12944‑017‑0444‑3.

  24. Murakami S, et al. Clin Exp Pharmacol Physiol. 2016;43(3):372‑378. doi:10.1111/1440‑1681.12534.

  25. Chen G. Curr Drug Targets. 2015;16(12):1281‑1292. doi:10.2174/1389450116666150325231015.

  26. Saeed A, et al. Nutrients. 2017;10(1):29. doi:10.3390/nu10010029.

  27. Xiang L, et al. Acta Biomater. 2023;168:497‑514. doi:10.1016/j.actbio.2023.07.035.

  28. Sun WL, et al. Nat Commun. 2023;14(1):477. doi:10.1038/s41467‑023‑36079‑x.

  29. Shimada M, et al. Nutr Res. 2019;64:49‑55. doi:10.1016/j.nutres.2019.01.002.

  30. Foster SR, et al. Chem Biol Interact. 2017;275:108‑115. doi:10.1016/j.cbi.2017.07.024.

 
 
 

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